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Please use this identifier to cite or link to this item: https://www.um.edu.mt/library/oar/handle/123456789/120235
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dc.contributor.authorGatt, Gianluca-
dc.date.accessioned2024-03-25T09:33:31Z-
dc.date.available2024-03-25T09:33:31Z-
dc.date.issued2022-
dc.identifier.citationGatt, G. (2022). The role of nitric oxide in stroke pathophysiology. Minima Medica, 2022, 66-73.en_GB
dc.identifier.urihttps://www.um.edu.mt/library/oar/handle/123456789/120235-
dc.description.abstractStroke is one of the leading causes of death and disability worldwide. Strokes can be classified as being either ischaemic strokes (IS) or haemorrhagic stokes (HS), mini strokes or transient ischaemic attacks (TIA). lschaemic strokes tend to be the most frequent, with an incidence of about 60-70% (1). Nitric oxide (NO) has been indicated to contribute to the pathogenesis of both ischaemic and haemorrhagic forms of stroke. [excerpt]en_GB
dc.language.isoenen_GB
dc.publisherMMSAen_GB
dc.rightsinfo:eu-repo/semantics/openAccessen_GB
dc.subjectCerebrovascular disease -- Pathophysiologyen_GB
dc.subjectStrokeen_GB
dc.subjectNitric oxide -- Physiological effecten_GB
dc.subjectIschemic strokeen_GB
dc.subjectNitric oxide -- Pathophysiologyen_GB
dc.titleThe role of nitric oxide in stroke pathophysiologyen_GB
dc.typearticleen_GB
dc.rights.holderThe copyright of this work belongs to the author(s)/publisher. The rights of this work are as defined by the appropriate Copyright Legislation or as modified by any successive legislation. Users may access this work and can make use of the information contained in accordance with the Copyright Legislation provided that the author must be properly acknowledged. Further distribution or reproduction in any format is prohibited without the prior permission of the copyright holder.en_GB
dc.description.reviewedpeer-revieweden_GB
dc.publication.titleMinima Medicaen_GB
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